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psx:substance/4-aco-det/claim/6be8205c-26bf-41cf-b926-d77b10bed4bb

claim2026-07-17recordsupported

derived from · upstream

feeds · downstream

Closure of psx:substance/4-aco-det/claim/6be8205c-26bf-41cf-b926-d77b10bed4bb: 73 upstream, 91 downstream.

psx:substance/4-aco-det/claim/6be8205c-26bf-41cf-b926-d77b10bed4bb
├─ used  src/doi:10.1007/s00204-018-2244-6  Opioid analgesic drugs and serotonin toxicity (syndrome): mechanisms, animal models, and links to clinical effects
  ├─ informed_by  claim/6b10666f-8df6-4726-833c-ef6b77a99540  Claim in safety — inconclusive  working
  ├─ informed_by  claim/261c5600-e413-4381-9e85-98b0e5a027a8  Claim in safety — inconclusive  working
  ├─ informed_by  claim/c08b8995-b126-4f2c-9ae9-31bf775a1147  Claim in safety — supported
  ├─ informed_by  claim/746f59da-a735-4016-9e12-5b0f823ee67c  Claim in safety — supported
  ├─ informed_by  claim/a97b95d8-9310-4d8e-96ab-fb729c3bba67  Claim in safety — inconclusive  working
  ├─ informed_by  claim/e507a8dc-01d5-47d5-a777-e6826aa56e9d  Claim in safety — supported
  ├─ informed_by  claim/6a7f0ab4-dd78-4b05-80e0-161f15a8018e  Claim in safety — supported
  ├─ informed_by  claim/b627739c-a878-4262-bb99-9a3f270481c6  Claim in safety — supported
  ├─ informed_by  claim/826f4d4f-aa65-4820-9431-118e00884b80  Claim in safety — inconclusive  working
  ├─ informed_by  claim/fb188a00-7567-4232-9a67-26b2761894b9  Claim in safety — supported
  ├─ informed_by  claim/06000273-cde8-467b-84d9-ebde70828449  Claim in safety — inconclusive  working
  ├─ informed_by  claim/0a6abcc0-b3f3-4c8d-b97a-b9346994e611  Claim in safety — inconclusive  working
  ├─ informed_by  claim/518eaf9e-b655-4f80-8d2d-3e85a27d7344  Claim in safety — supported
  ├─ informed_by  claim/a87c7221-a74b-4335-b259-3818f17f62ec  Claim in safety — supported
  ├─ informed_by  claim/5734f5c7-23b5-4c21-ab0e-0a3b3366ef78  Claim in safety — partially_supported
  ├─ informed_by  claim/0e31ab2e-821a-4fbe-ab19-413309795dfc  Claim in safety — supported
  ├─ informed_by  claim/9477211a-e688-421f-9c7c-91e68ecfac4b  Claim in safety — supported
  ├─ informed_by  claim/68e94451-0ec4-48d8-803d-af5ccc8bced3  Claim in safety — supported
  ├─ informed_by  claim/5b52f8f0-2617-4e87-8916-859231cc1558  Claim in safety — partially_supported
  ├─ informed_by  claim/362bb9fb-b61b-4ee6-b648-5b326a2e9b43  Claim in safety — supported
    ├─ used  src/url:a9a7a4460a42dd90  4-Acetoxy-DET (Ethacetin) - Wikidoc
    ├─ used  src/doi:10.3389/fpsyt.2022.1020505  Lower-Dose Psycholytic Therapy - A Neglected Approach
    ├─ used  src/url:4cc1e24bda32b779  4-AcO-DET/Ethacetin Degradation (Erowid)
    ├─ used  src/doi:10.1021/acs.jmedchem.3c01225  Novel Psilocin Prodrugs with Altered Pharmacological Properties as Candidate Therapies for Treatment-Resistant Anxiety Disorders
    ├─ used  src/pmid:2139186  Differential interactions of indolealkylamines with 5-hydroxytryptamine receptor subtypes
    ├─ used  src/doi:10.1021/acschemneuro.2c00170  Tolerance and Cross-Tolerance among Psychedelic and Nonpsychedelic 5-HT2A Receptor Agonists in Mice
    ├─ used  src/url:b18ca63773199877  4-HO-DET (CZ-74) — DMT-Nexus Wiki
    ├─ used  src/url:c0d1fa9c049ba571  Hanscarl Leuner: Pioneer of Hallucinogen Research and Psycholytic Therapy
    ├─ used  src/url:c843cbe335da96c8  DEA Scheduling: PMA as Schedule I hallucinogen (38 FR 26447, July 2, 1973)
    ├─ used  src/url:bf5b46070f6f77a2  Schedules of Controlled Substances: Proposed Placement of 4-OH-DiPT, 5-MeO-AMT, 5-MeO-MiPT, 5-MeO-DET, and DiPT in Schedule I
    ├─ used  src/url:47d86a78d170c66b  4-AcO-DET (Ethacetin) compound record
    ├─ used  src/doi:10.16889/isomerdesign-5-supp  PeakAL: Protons I Have Known and Loved, Too - Another Fifty Shades of Grey-Market Spectra. Supplementary Data
    ├─ used  src/doi:10.1124/jpet.122.001454  Pharmacologic Activity of Substituted Tryptamines at 5-HT2A, 5-HT2C, 5-HT1A Receptors and SERT
    ├─ used  src/doi:10.3390/pharmaceutics17040411  Pharmacokinetics of Psilocybin: A Systematic Review
    ├─ used  src/doi:10.1523/jneurosci.1384-23.2023  Beyond the 5-HT2A Receptor: Classic and Nonclassic Targets in Psychedelic Drug Action
    ├─ used  src/doi:10.1021/acsptsci.2c00222  Receptor Binding Profiles for Tryptamine Psychedelics and Effects of 4-Propionoxy-N,N-dimethyltryptamine in Mice
    ├─ used  src/doi:10.1021/acsptsci.2c00177  Structure-Activity Relationships for Psilocybin, Baeocystin, Aeruginascin, and Related Analogues to Produce Pharmacological Effects in Mice
    ├─ used  src/doi:10.1016/j.neuropharm.2019.107933  Correlation between the potency of hallucinogens in the mouse head-twitch response assay and their behavioral and subjective effects in other species
    ├─ used  src/doi:10.1016/j.euroneuro.2016.05.001  Receptor interaction profiles of novel psychoactive tryptamines compared with classic hallucinogens
    ├─ used  src/doi:10.2174/1570159X13666141210222409
    ├─ used  src/doi:10.3390/ijms21239279  Toxicology and Analysis of Psychoactive Tryptamines.
    ├─ used  src/doi:10.1124/jpet.111.179705  Abuse liability profile of three substituted tryptamines
    ├─ used  src/doi:10.1345/aph.1Q748
    ├─ used  src/doi:10.1080/15563650.2021.1955912  Marked and prolonged serotonin toxicity in a tramadol-poisoned patient with a pharmacokinetic study
    ├─ used  src/pmid:11101361
    ├─ used  src/pmid:17905422
    ├─ used  src/doi:10.1007/s00213-014-3658-3
    ├─ used  src/doi:10.3109/00365513.2013.793817
    ├─ used  src/doi:10.1007/s00213-022-06279-2
    ├─ used  src/url:c4d2b006b99cb364
    ├─ derived_from  gen/eb5c61ff-1cd1-48a7-a296-e65614085d33  Generation — pipeline-v5
    ├─ generated_by  pub/95d5fdce-bbb7-43da-b21b-830efc0b72cb  Publication — backfill
      ├─ used  search/dbb37bef-2384-40be-9973-ad32608d05a5  Searched PubChem for "4-AcO-DET"
      ├─ used  search/4535d671-f6bc-477f-8c68-4f4bd14c9b55  Searched PubMed for "4-AcO-DET ethacetin diethyltryptamine acetate pharmacology"
      ├─ used  search/7850d349-6d33-4bad-8db9-b75e34168d5a  Searched OpenAlex for "4-acetoxy-DET ethacetin tryptamine psychedelic"
      ├─ used  search/1eab1057-0efa-4b6e-9b07-d5e58d27466e  Searched ClinicalTrials.gov for "4-AcO-DET ethacetin"
      ├─ used  search/c1f31082-fdde-48d7-88d7-ae6c7d91bcea  Searched PubMed for "substituted tryptamine serotonin 5-HT2A receptor binding affinity psychedelic"
      ├─ used  search/3b946f38-6a50-44ca-b94b-484eb1c4f2ed  Searched OpenAlex for "diethyltryptamine DET psychedelic serotonin receptor"
      ├─ used  search/1ab568aa-2605-4479-8947-90e14d194267  Searched PubMed for "4-acetoxy tryptamine psilocin prodrug serotonin receptor"
      ├─ used  search/f98221d0-2bf3-4bc0-b115-f20739b54a40  Searched PubMed for "tryptamine serotonin syndrome drug interaction tramadol"
      ├─ used  search/2c2c9c32-2de7-413c-9e5f-2e1163c687de  Searched PubMed for "tryptamine psychedelic cross tolerance serotonin 5-HT2A receptor downregulation"
      ├─ used  search/f241f7bb-165c-44c8-8f16-8b195bb58c39  Searched PsychonautWiki for "4-AcO-DET pharmacology dosage"
      ├─ used  search/f2b10ada-d350-4e47-8f34-4d35b8e38bb8  Searched Wikidoc for "4-AcO-DET history synthesis legal status"
      ├─ used  search/d43766a0-4cb1-47ea-a1ed-81fb8fe4e5da  Searched Erowid for "4-AcO-DET stability degradation"
      ├─ used  search/0868f4cf-49ce-4f92-9c28-8938c0e05775  Searched Web for "4-AcO-DET UK legal status scheduling tryptamine"
      ├─ used  search/10b37cde-dddc-44aa-b132-0a5baf9dfac9  Searched PubMed for "psilocybin psilocin pharmacokinetics CYP metabolism human"
      ├─ used  search/0a29e2b6-5391-45c1-a0a3-4b4e9353f2c3  Searched PubMed for "psychedelic tryptamine head twitch drug discrimination rat mouse"
      ├─ used  search/f9461b69-a46d-41ce-82a5-922742eccc4c  Searched OpenAlex for "4-acetoxy tryptamine prodrug psilocin 5-HT2A receptor"
      ├─ used  search/6acfe90c-e723-4cfc-874d-b9e146c147be  Searched MAPS/web for "Leuner CZ-74 clinical psycholytic therapy"
      ├─ used  search/5fcd3278-b54f-446a-b142-1d090d9667c0  Searched Web/GOV.UK for "UK Psychoactive Substances Act 2016 tryptamine"
      ├─ used  search/cd6875e4-1eb0-42d4-a333-dbdf2509ecea  Searched PubMed for "novel psychoactive substance tryptamine intoxication emergency department"
      └─ used  search/a7ac1187-867b-41cd-9fbe-59b0aa5a0792  Searched PubChem/Wikidoc/Erowid for "4-AcO-DET chemical identity cross-reference"
├─ informed_by  section/safety.full@1
  ├─ used  version/1  Revision 1
  ├─ used  claim/a9a17030-4aeb-4677-9bac-66421b33ee7f  Claim in safety — supported
  ├─ used  claim/0b4d681a-e5d3-4169-aeb3-c5e3f8e1e7bb  Claim in safety — supported
  ├─ used  claim/f549f62c-8254-402d-a036-561c1cfa9b62  Claim in safety — supported
    ├─ derived_from  issue/notice@64878b43a1d2  Issued notice
    ├─ derived_from  issue/info_card@e287cd50add8  Issued info_card
    ├─ derived_from  issue/emergency@3f0909c25c71  Issued emergency
    ├─ derived_from  issue/notice@f069a9dea47b  Issued notice
    ├─ derived_from  issue/info_card@6384cb478828  Issued info_card
    ├─ derived_from  issue/emergency@c15c760aabf6  Issued emergency
      ├─ used  claim/35b68d48-ab8e-47ed-b255-4fb2f5554e3b  Claim in summary — inconclusive  working
      ├─ used  claim/aaa4da7a-0fa5-42ff-82f8-1d09c04475b9  Claim in history — supported
      ├─ used  claim/b18f3ddf-ea81-4a3d-9e6e-19dfa88f6c96  Claim in history — supported
      ├─ used  claim/4dad1bd0-b342-4c68-b180-73f3049434d6  Claim in clinical-evidence — supported
      ├─ used  claim/8054ab5b-64dc-4a97-9677-df6f99279aa0  Claim in effects-overview — inconclusive  working
      ├─ used  claim/ff147b95-b227-4a3d-800a-6ce678bdba8a  Claim in summary — supported
      ├─ used  claim/4789ec70-6b5a-4b1a-b935-6b2f15236a9b  Claim in history — supported
      ├─ used  claim/1c5abbe0-193e-4afc-a300-6cfb4c192735  Claim in history — supported
      ├─ used  claim/6182136c-361b-448b-b1a9-9af0e848e1f2  Claim in clinical-evidence — supported
      ├─ used  claim/3779048e-be21-42a9-b633-ded95923749d  Claim in effects-overview — supported
      ├─ used  claim/84ed32c6-c3cf-4bd9-ad5c-8b0feb5b55d0  Claim in summary — supported
      ├─ used  claim/0aa381e4-e9d1-43d7-b3c6-e1a5271264e5  Claim in pharmacology — supported
      ├─ used  claim/737a5b25-a846-4ac9-85a3-0dc223e4cbe5  Claim in pharmacology — partially_supported
      ├─ used  claim/1a9c189b-e3ed-4c50-83ff-2063120fff38  Claim in pharmacology — supported
      ├─ used  claim/a17dee18-0f54-40b9-9fbd-357bb254bc99  Claim in chemistry — supported
      ├─ used  claim/b137d7f2-6061-49d1-a705-e914c34b7759  Claim in pharmacokinetic-detail — supported
      ├─ used  claim/76dd723f-1e46-41d2-8c5f-f2defaffcfcd  Claim in comparative-pharmacology — supported
      ├─ used  claim/ef376caf-4bbe-4fb1-a639-995918efcf78  Claim in summary — supported
      ├─ used  claim/1c5669ae-7b7e-46f2-9d70-db492da3129f  Claim in pharmacology — supported
      ├─ used  claim/9d7d6820-d54e-4d06-9a15-c0e2832c9687  Claim in pharmacology — partially_supported
      ├─ used  claim/ed443aaa-4957-4af5-bb7b-990111bd2952  Claim in pharmacology — supported
      ├─ used  claim/f9898dac-51b4-417e-a29b-7b4516cb2f0f  Claim in chemistry — supported
      ├─ used  claim/5dfbdf06-2679-4070-9397-45d4f72a3ecb  Claim in pharmacokinetic-detail — supported
      ├─ used  claim/9e3289e5-da4d-4067-8cb1-4978164e16ac  Claim in comparative-pharmacology — supported
      ├─ used  claim/8143a705-16cc-492d-9f20-0649e5e421e8  Claim in summary — supported
      ├─ used  claim/8107dd98-9402-42fa-b38f-4408928f5810  Claim in pharmacology — partially_supported
      ├─ used  claim/a8d36113-e6e8-49c2-b8a3-1bd44ea8bcf2  Claim in chemistry — supported
      ├─ used  claim/33ea9d8c-38df-4c29-8bb5-8483645d17ee  Claim in receptor-pharmacology — supported
      ├─ used  claim/914053fa-5b6d-44bc-bc8f-656f07fef65a  Claim in comparative-pharmacology — supported
      ├─ used  claim/1b653a14-8f98-4aeb-b96b-1d486d5e1942  Claim in summary — supported
      ├─ used  claim/b524bee1-fc83-4317-9f7b-3825dccb8cea  Claim in pharmacology — partially_supported
      ├─ used  claim/44ecaab2-c3ff-402c-a28f-d7b4183e3e3d  Claim in chemistry — supported
      ├─ used  claim/7e09d178-328a-4e3e-aa7c-2bab1e90e294  Claim in receptor-pharmacology — supported
      ├─ used  claim/5bac7559-3b7e-4fcd-9549-73438aed515b  Claim in comparative-pharmacology — supported
      ├─ used  claim/cfd72ffa-08bc-4641-bbed-f05e08484922  Claim in summary — partially_supported
      ├─ used  claim/10b25c41-9453-426f-bf55-4c501f7c93d3  Claim in summary — partially_supported
      ├─ used  claim/f8d7a887-bc23-4550-adcf-5d26985069bf  Claim in chemistry — supported
      ├─ used  claim/d070d6c2-9d5f-4544-a22c-49cc0e7efbd0  Claim in chemistry — supported
      ├─ used  claim/6d26e1a0-4292-4fc6-96e6-705b4008a084  Claim in pharmacology — supported
      ├─ used  claim/9d6d1e34-4c12-4197-8ba7-671b46839463  Claim in receptor-pharmacology — supported
      ├─ used  claim/9f95ecbc-c1ba-4762-ad5d-b71501d0c676  Claim in receptor-pharmacology — supported
      ├─ used  claim/33b25e49-4c44-4738-866c-37c6feb37bf9  Claim in receptor-pharmacology — supported
      ├─ used  claim/c9b00173-1943-4825-a387-8d27c27fabf1  Claim in pharmacology — supported
      ├─ used  claim/03efa009-5568-4d69-bc68-022859e0a367  Claim in receptor-pharmacology — supported
      ├─ used  claim/597a6956-dd7e-4a48-9597-5bf54cdd0153  Claim in receptor-pharmacology — inconclusive  working
      ├─ used  claim/6d04ac1b-0935-4a19-8d82-d25aa17d08c2  Claim in receptor-pharmacology — supported
      ├─ used  claim/bc9abed5-7837-45c6-97b8-3848cc402187  Claim in pharmacology — supported
      ├─ used  claim/dd6793f8-f79b-4c14-88f1-3b87b0262aef  Claim in pharmacology — supported
      ├─ used  claim/4f3f3d6c-3d59-45dc-b87c-64b4fc80c9ae  Claim in pharmacokinetic-detail — supported
      ├─ used  claim/551ed6b8-8124-4a24-a9e6-e32ab85b68d6  Claim in pharmacokinetic-detail — partially_supported
      ├─ used  claim/4c416bdc-6f39-4802-baf1-1cceae58f364  Claim in pharmacology — supported
      ├─ used  claim/e851ac67-25df-4040-9e31-300ebf2ae0b0  Claim in pharmacology — supported
      ├─ used  claim/1f8b4a33-55e6-486b-b585-3d1c27093ea3  Claim in pharmacokinetic-detail — supported
      ├─ used  claim/4fc5ebd6-ff5c-46de-a249-1e8ef561c939  Claim in pharmacokinetic-detail — partially_supported
      ├─ used  claim/122d8f43-c202-489f-a009-4a46fe8540b7  Claim in receptor-pharmacology — supported
      ├─ used  claim/edb786c4-dd1b-4aa1-a5ac-9f5e769d9665  Claim in receptor-pharmacology — supported
      ├─ used  claim/4a05bca4-b1f8-4526-98ce-ecce918f5e8d  Claim in neuroscience — supported
      ├─ used  claim/19e592e6-5a8e-44ec-894c-c0afb6e2ee61  Claim in neuroscience — inconclusive  working
      ├─ used  claim/14b2d67d-0137-499a-8fa9-db58aaca4719  Claim in receptor-pharmacology — supported
      ├─ used  claim/bd17c024-8de7-4062-a889-1fcd3cd43ca8  Claim in receptor-pharmacology — supported
      ├─ used  claim/44582b2f-55cb-4673-ab5b-475cae4deae3  Claim in neuroscience — supported
      ├─ used  claim/53e1acec-25c0-4b46-b797-9954207e51cb  Claim in neuroscience — supported
      ├─ used  claim/0a6e6552-d4c7-41c7-b908-bade5743c479  Claim in receptor-pharmacology — supported
      ├─ used  claim/248d3945-db2b-4cd2-a618-a8d4adb398f0  Claim in receptor-pharmacology — supported
      ├─ used  claim/d3858af1-f969-4816-bb39-4c74e9a790f5  Claim in receptor-pharmacology — supported
      ├─ used  claim/72fcca9d-aa15-4b66-a5c5-15030e27d2da  Claim in comparative-pharmacology — supported
      ├─ used  claim/15264776-3202-4fb7-98c1-d552f61ea24b  Claim in receptor-pharmacology — supported
      ├─ used  claim/9c43c65d-618a-4dfb-b4e5-278ecb531926  Claim in receptor-pharmacology — supported
      ├─ used  claim/a5813437-5669-4446-ad23-599223b459cd  Claim in receptor-pharmacology — supported
      ├─ used  claim/b827e508-e340-47dc-952a-3a7a97c22ac1  Claim in comparative-pharmacology — inconclusive  working
      ├─ used  claim/10cbea0a-9390-4bf3-8925-409b44e8f34f  Claim in receptor-pharmacology — inconclusive  working
      ├─ used  claim/d0a411c4-a4a9-47dc-a7a3-7adae7959d39  Claim in comparative-pharmacology — supported
      ├─ used  claim/58b3c85d-d559-4820-b1f1-df82d33c1568  Claim in receptor-pharmacology — partially_supported
      ├─ used  claim/d65d5f61-eae6-43f4-b070-8f6df3d4009e  Claim in comparative-pharmacology — inconclusive  working
      ├─ used  claim/16c872ad-01dd-4464-bbd1-46323142e43c  Claim in receptor-pharmacology — inconclusive  working
      ├─ used  claim/47ef6253-bbc5-4038-8e40-327e0fbf59af  Claim in receptor-pharmacology — inconclusive  working
      ├─ used  claim/a025e515-d17b-41c0-b4d3-89acc438f113  Claim in receptor-pharmacology — inconclusive  working
      ├─ used  claim/242289b3-352e-46f6-b08f-58827f7991bc  Claim in receptor-pharmacology — supported
      ├─ used  claim/5c402d78-9ad8-4a52-80a6-699bcd84fcea  Claim in receptor-pharmacology — inconclusive  working
      └─ used  claim/d142e4de-379c-47a4-b7f4-cf60d63d6cf2  Claim in receptor-pharmacology — supported
… closure truncated at 200 nodes

indented by distance from this node, not by parentage

1Provenance card

the boundary, as counts

Sources

2517 with DOI

4 strong · 14 moderate · 7 weak

Compilations

12026-04-28T03:58:35.187Z

pipeline-v5 · prompt hash not recorded — run predates 2026-09

Decisions

30last ruled 2026-06-04

0 awaiting a ruling

Issued documents

62026-10-19T20:29:48.212Z

notice · info card · emergency

Searches

203 with no results

queries and counts on the build

Claims checked

10487 supported

87 supported · 17 not

Correspondence

0received

consent field ships in Phase 4

Edges

306

derivations recorded for this entity

Complete over 25 sources, 1 compilation, and 30 decisions.Not here: the private journal (never).Working material — drafts, discards, unsupported claims — is listed below and readable on the working page.Cite: psychedex.org/substances/4-aco-det/archive

2Build

one panel per compilation run

generation run 2026-04-28

24,852 min completedmodel pipeline-v5plan not recordedrun/bfab76b1…

Selection

results seen77
selected by the model32
consumed as sources25

Verification

claims checked104
supported76
partially supported11
not supported0
inconclusive17
  • record
  • seen / partial
  • working

The model's own caveats

  • no_substance_specific_binding_data

Reproduce

inputs
25 source keys (listed under Sources)
plan
not recorded — template version, prompt hash and git SHA are written from 2026-09 onward
model
pipeline-v5
streams
none — agent transcripts are persisted from 2026-09 onward
20 searches — query, source, results, what was kept and why
20 searches for this run
#sourcequeryseenkeptrationale
1PubMed4-acetoxy tryptamine psilocin prodrug serotonin receptor11—
2ClinicalTrials.gov · absence4-AcO-DET ethacetin0——
3PubMedtryptamine psychedelic cross tolerance serotonin 5-HT2A receptor downregulation11—
4OpenAlexdiethyltryptamine DET psychedelic serotonin receptor104—
5PubMed · absence4-AcO-DET ethacetin diethyltryptamine acetate pharmacology0——
6OpenAlex · absence4-acetoxy-DET ethacetin tryptamine psychedelic0——
7PubMedsubstituted tryptamine serotonin 5-HT2A receptor binding affinity psychedelic154—
8PubChem4-AcO-DET11Only result; confirms chemical identity data from seed brief.
9PubMedtryptamine serotonin syndrome drug interaction tramadol82—
10Web4-AcO-DET UK legal status scheduling tryptamine103—
11PubMedpsychedelic tryptamine head twitch drug discrimination rat mouse43—
12PubMedpsilocybin psilocin pharmacokinetics CYP metabolism human11—
13Erowid4-AcO-DET stability degradation13Key stability and market history data.
14PsychonautWiki4-AcO-DET pharmacology dosage1—Cross-reference for dose data.
15Wikidoc4-AcO-DET history synthesis legal status13Primary historical data about 4-AcO-DET first synthesis.
16Web/GOV.UKUK Psychoactive Substances Act 2016 tryptamine102—
17MAPS/webLeuner CZ-74 clinical psycholytic therapy1—Historical context for CZ-74/4-HO-DET clinical use.
18PubChem/Wikidoc/Erowid4-AcO-DET chemical identity cross-reference3—Verifying all chemical identity data against primary source.
19PubMednovel psychoactive substance tryptamine intoxication emergency department11—
20OpenAlex4-acetoxy tryptamine prodrug psilocin 5-HT2A receptor83—
104 claims by section — every cited sentence, checked against its source
receptor-pharmacology26 · 20 ok
  • The primary psychedelic mechanism is 5-ht2a receptor agonismsupported

    4-substituted compounds show high selectivity for 5-HT2AR. jpet.122.001454

  • The primary psychedelic mechanism is 5-ht2a receptor agonismsupported

    Review of psychedelic mechanisms beyond 5-HT2A including 5-HT1A, 5-HT2C, TrkB receptors. jneurosci.1384-23.2023

  • Kozell et al. (2023) characterized 21 substituted tryptamines at 5-ht1a, 5-HT₂A, 5-ht2c, and serotonin-transporter using human-receptor-expressing HEK cells. 4-Substituted tryptamines exhibited high selectivity for 5-HT₂A over 5-HT₁A and 5-HT₂C, with affinities spanning a 100-fold range across compounds tested, including 4-acetoxy derivatives.supported

    21 substituted tryptamines at 5-HT1A, 5-HT2A, 5-HT2C and SERT. 4-substituted compounds show high selectivity for 5-HT2AR. jpet.122.001454

  • Glatfelter et al. (2023) determined receptor binding profiles for tryptamines with 4-position substituents (hydroxy, acetoxy, propionoxy) and N,N-dialkyl substitutions including N,N-diethyl — directly relevant to 4-AcO-DETsupported

    4-Acetoxy and 4-propionoxy analogues of 4-hydroxy compounds displayed somewhat weaker binding affinities but similar target profiles across 5-HT receptors and other identified targets. acsptsci.2c00222

  • McKenna et al. (1990) established that 4-hydroxylated indolealkylamines have 25–380-fold selectivity for the 5-HT₂A site over 5-HT₁Asupported

    4-hydroxylated derivatives show 25-380-fold selectivity for 5-HT2A vs 5-HT1A. pmid:2139186

  • Claim in receptor-pharmacology — inconclusiveinconclusiveworking →

  • Claim in receptor-pharmacology — inconclusiveinconclusiveworking →

  • Claim in receptor-pharmacology — inconclusiveinconclusiveworking →

  • Secondary agonist activity at 5-HT₁A receptors is documented for the 4-substituted tryptamine classsupported

    The tryptamine psychedelics target multiple serotonin receptors, including 5-HT1A. acsptsci.2c00222

  • At high doses, 5-HT₁A activation attenuates the 5-HT₂A-mediated head twitch response and produces hypothermia and hypolocomotion in mice.supported

    4-PrO-DMT displayed dose-related psilocybin-like effects in mice: 5-HT2A-mediated HTR (0.3–3 mg/kg s.c.) and 5-HT1A-mediated hypothermia and hypolocomotion (3–30 mg/kg s.c.). acsptsci.2c00222

  • Some affinity at 5-HT₂C is present for 4-substituted compounds, but lower than at 5-HT₂A.supported

    21 substituted tryptamines at 5-HT1A, 5-HT2A, 5-HT2C and SERT. 4-substituted compounds show high selectivity for 5-HT2AR. jpet.122.001454

  • Rickli et al. (2016) found that tryptamines including 4-OH-dipt — the closest structurally characterized analog to 4-HO-DET — interacted with the serotonin transporter (SERT) similarly to mdmasupported

    Mescaline does not interact with serotonin, norepinephrine, or dopamine transporters — in contrast to MDMA and several tryptamines. j.euroneuro.2016.05.001

  • Serotonergic psychedelics also interact with TrkB neurotrophin receptors, which may contribute to neuroplasticity effectssupported

    Review of psychedelic mechanisms beyond 5-HT2A including 5-HT1A, 5-HT2C, TrkB receptors. Neuroplasticity promotion reviewed. jneurosci.1384-23.2023

  • The primary psychedelic mechanism is 5-ht2a receptor agonismsupported

    4-substituted compounds show high selectivity for 5-HT2AR. jpet.122.001454

  • The primary psychedelic mechanism is 5-ht2a receptor agonismsupported

    Review of psychedelic mechanisms beyond 5-HT2A including 5-HT1A, 5-HT2C, TrkB receptors. jneurosci.1384-23.2023

  • Claim in receptor-pharmacology — inconclusiveinconclusiveworking →

  • Glatfelter et al. (2023) determined receptor binding profiles for tryptamines with 4-position substituents (hydroxy, acetoxy, propionoxy) and N,N-dialkyl substitutions including N,N-diethyl — directly relevant to 4-AcO-DETsupported

    4-Acetoxy and 4-propionoxy analogues of 4-hydroxy compounds displayed somewhat weaker binding affinities but similar target profiles across 5-HT receptors and other identified targets. acsptsci.2c00222

  • McKenna et al. (1990) established that 4-hydroxylated indolealkylamines have 25–380-fold selectivity for the 5-HT₂A site over 5-HT₁Asupported

    4-hydroxylated derivatives show 25-380-fold selectivity for 5-HT2A vs 5-HT1A. pmid:2139186

  • Glatfelter et al. (2022) demonstrated in mouse models that 4-acetoxy tryptamines display nanomolar affinity for most human 5-HT receptor subtypes, including 5-HT₂A and 5-HT₁Apartially supported

    SAR for psilocybin and analogs. 4-Acetoxy and 4-hydroxy compounds showed similar pharmacological effects. acsptsci.2c00177

  • Claim in receptor-pharmacology — inconclusiveinconclusiveworking →

  • Claim in receptor-pharmacology — inconclusiveinconclusiveworking →

  • Secondary agonist activity at 5-HT₁A receptors is documented for the 4-substituted tryptamine classsupported

    The tryptamine psychedelics target multiple serotonin receptors, including 5-HT1A. 4-PrO-DMT displayed dose-related psilocybin-like effects in mice: 5-HT2A-mediated HTR (0.3–3 mg/kg s.c.) and 5-HT1A-mediated hypothermia and hypolocomotion (3–30 mg/kg s.c.). acsptsci.2c00222

  • At high doses, 5-HT₁A activation attenuates the 5-HT₂A-mediated head twitch response and produces hypothermia and hypolocomotion in mice.supported

    4-PrO-DMT displayed dose-related psilocybin-like effects in mice: 5-HT2A-mediated HTR (0.3–3 mg/kg s.c.) and 5-HT1A-mediated hypothermia and hypolocomotion (3–30 mg/kg s.c.). acsptsci.2c00222

  • Some affinity at 5-HT₂C is present for 4-substituted compounds, but lower than at 5-HT₂A.supported

    21 substituted tryptamines at 5-HT1A, 5-HT2A, 5-HT2C and SERT. 4-substituted compounds show high selectivity for 5-HT2AR. jpet.122.001454

  • Rickli et al. (2016) found that tryptamines including 4-OH-dipt — the closest structurally characterized analog to 4-HO-DET — interacted with the serotonin transporter (SERT) similarly to mdmasupported

    Mescaline does not interact with serotonin, norepinephrine, or dopamine transporters — in contrast to MDMA and several tryptamines. j.euroneuro.2016.05.001

  • Serotonergic psychedelics also interact with TrkB neurotrophin receptors, which may contribute to neuroplasticity effectssupported

    Review of psychedelic mechanisms beyond 5-HT2A including 5-HT1A, 5-HT2C, TrkB receptors. Neuroplasticity promotion reviewed. jneurosci.1384-23.2023

safety24 · 18 ok
  • Claim in safety — inconclusiveinconclusiveworking →

  • Claim in safety — inconclusiveinconclusiveworking →

  • This absence may reflect limited clinical detection rather than true absence of adverse events: Malaca et al. (2020) noted that tryptamine contribution to drug overdoses 'may be underestimated' because these compounds are not included in standard toxicology screening panels.supported

    morbidity considerable and underestimated. ijms21239279

  • De la Fuente Revenga et al. (2022) demonstrated in mice that rapid tolerance to 5-HT₂A-mediated head twitch response develops after a single dose, involves 5-HT₂A receptor downregulation in the frontal cortex, and produces cross-tolerance between psychedelics (doi and lsd were tested)supported

    Tolerance after single dose. Cross-tolerance between DOI and LSD. DOI caused 5-HT2AR downregulation in frontal cortex. acschemneuro.2c00170

  • Claim in safety — inconclusiveinconclusiveworking →

  • The active metabolite 4-HO-DET is a 5-HT₂A agonist that may also interact with serotonin-transporter, creating multiple mechanistic pathways for serotonin-syndrome in combination settings.supported

    Mescaline does not interact with serotonin, norepinephrine, or dopamine transporters — in contrast to MDMA and several tryptamines. j.euroneuro.2016.05.001

  • Tramadol poses the most clearly documented danger. tramadol inhibits SERT, and Baldo (2018) identified it as among the opioids most associated with serotonin syndrome in humanssupported

    Tramadol inhibits human SERT in HEK293 cells. Combined with serotonergic drugs (MAOIs, SSRIs), tramadol can precipitate serotonin syndrome. s00204-018-2244-6

  • Tramadol poses the most clearly documented danger. tramadol inhibits SERT, and Baldo (2018) identified it as among the opioids most associated with serotonin syndrome in humanssupported

    Tramadol inhibits serotonin reuptake and has opioid effects that compound serotonergic risk. aph.1q748

  • Tramadol poses the most clearly documented danger. tramadol inhibits SERT, and Baldo (2018) identified it as among the opioids most associated with serotonin syndrome in humanssupported

    Tramadol inhibits human SERT in HEK293 cells. Combined with serotonergic drugs (MAOIs, SSRIs), tramadol can precipitate serotonin syndrome. s00204-018-2244-6

  • Case reports confirm severe and prolonged serotonin toxicity from tramadolsupported

    Case report of severe and prolonged serotonin toxicity from tramadol. Pharmacokinetic study confirming mechanism. 15563650.2021.1955912

  • Claim in safety — inconclusiveinconclusiveworking →

  • Monoamine oxidase inhibitors (MAOIs) would be expected to dramatically potentiate effects by inhibiting mao-a-mediated metabolic inactivation of the active metabolite 4-HO-DETsupported

    Primary metabolism by CYP2D6 and CYP3A4; secondary MAO-A. pharmaceutics17040411

  • Claim in safety — inconclusiveinconclusiveworking →

  • Claim in safety — inconclusiveinconclusiveworking →

  • This absence may reflect limited clinical detection rather than true absence of adverse events: Malaca et al. (2020) noted that tryptamine contribution to drug overdoses "may be underestimated" because these compounds are not included in standard toxicology screening panels.supported

    morbidity considerable and underestimated. ijms21239279

  • De la Fuente Revenga et al. (2022) demonstrated in mice that rapid tolerance to 5-HT₂A-mediated head twitch response develops after a single dose, involves 5-HT₂A receptor downregulation in the frontal cortex, and produces cross-tolerance between psychedelics (doi and lsd were tested)supported

    Tolerance after single dose. Cross-tolerance between DOI and LSD. DOI caused 5-HT2AR downregulation in frontal cortex. acschemneuro.2c00170

  • Gatch et al. (2011) found low reinforcement and low abuse-liability for related substituted tryptamines in rodent self-administration paradigmspartially supported

    DOM used as training drug. DIPT fully substituted at ED50=1.94 mg/kg. Low abuse liability for DOM in rodents. jpet.111.179705

  • The active metabolite 4-HO-DET is a 5-HT₂A agonist that may also interact with serotonin-transporter, creating multiple mechanistic pathways for serotonin-syndrome in combination settings.supported

    Mescaline does not interact with serotonin, norepinephrine, or dopamine transporters — in contrast to MDMA and several tryptamines. j.euroneuro.2016.05.001

  • Tramadol poses the most clearly documented danger. tramadol inhibits SERT, and Baldo (2018) identified it as among the opioids most associated with serotonin syndrome in humanssupported

    Tramadol inhibits human SERT in HEK293 cells. Combined with serotonergic drugs (MAOIs, SSRIs), tramadol can precipitate serotonin syndrome. s00204-018-2244-6

  • Tramadol poses the most clearly documented danger. tramadol inhibits SERT, and Baldo (2018) identified it as among the opioids most associated with serotonin syndrome in humanssupported

    Tramadol inhibits serotonin reuptake and has opioid effects that compound serotonergic risk. aph.1q748

  • Tramadol poses the most clearly documented danger. tramadol inhibits SERT, and Baldo (2018) identified it as among the opioids most associated with serotonin syndrome in humanssupported

    Tramadol inhibits human SERT in HEK293 cells. Combined with serotonergic drugs (MAOIs, SSRIs), tramadol can precipitate serotonin syndrome. s00204-018-2244-6

  • Case reports confirm severe and prolonged serotonin toxicity from tramadolsupported

    Case report of severe and prolonged serotonin toxicity from tramadol. Pharmacokinetic study confirming mechanism. 15563650.2021.1955912

  • Case reports confirm severe and prolonged serotonin toxicity from tramadolpartially supported

    Reviews the pharmacodynamic basis of tramadol-SSRI serotonin syndrome interactions. Tramadol inhibits serotonin reuptake and has opioid effects that compound serotonergic risk. aph.1q748

  • Monoamine oxidase inhibitors (MAOIs) would be expected to dramatically potentiate effects by inhibiting mao-a-mediated metabolic inactivation of the active metabolite 4-HO-DETsupported

    Primary metabolism by CYP2D6 and CYP3A4; secondary MAO-A. pharmaceutics17040411

pharmacology14 · 14 ok
  • 4-AcO-DET is a presumed prodrug that undergoes hydrolysis via serum esterases to yield 4-HO-DET (ethocin/CZ-74), the pharmacologically active speciessupported

    Screened 28 novel psilocin prodrugs using 4-position cleavable groups including acetoxy. 4-acetoxy substitution modulates metabolic processing and pharmacokinetic profile. acs.jmedchem.3c01225

  • Raithatha et al. (2024) demonstrated that 4-acetoxy groups on indole tryptamines function as cleavable prodrug moieties generating the active 4-hydroxy form, but noted that 4-acetoxy compounds show 'somewhat weaker binding affinities but similar target profiles across serotonin receptors' compared to their 4-hydroxy counterparts.partially supported

    Novel prodrugs showed reduced pharmacological exposure vs psilocybin while retaining psilocin activity. acs.jmedchem.3c01225

  • Class-level data establish that 4-substituted tryptamines exhibit nanomolar 5-ht2a affinity with 25–380-fold selectivity over 5-ht1a and greater selectivity over 5-ht2c.partially supported

    Binding affinities of 21 indolealkylamine derivatives at 5-HT1A, 5-HT2A, 5-HT2B by radioligand competition. 4-hydroxylated derivatives show 25-380-fold selectivity for 5-HT2A vs 5-HT1A. pmid:2139186

  • Class-level data establish that 4-substituted tryptamines exhibit nanomolar 5-ht2a affinity with 25–380-fold selectivity over 5-ht1a and greater selectivity over 5-ht2c.supported

    21 substituted tryptamines at 5-HT1A, 5-HT2A, 5-HT2C and SERT. 4-substituted compounds show high selectivity for 5-HT2AR. jpet.122.001454

  • Raithatha et al. (2024) found that 4-acetoxy prodrugs demonstrated 'reduced pharmacological exposure compared with psilocybin,' suggesting slower or less complete activation than phosphate ester prodrugs.supported

    Novel prodrugs showed reduced pharmacological exposure vs psilocybin while retaining psilocin activity. acs.jmedchem.3c01225

  • By analogy to Psilocin (4-HO-dmt), the active metabolite 4-HO-DET is expected to be metabolized primarily by CYP2D6 and CYP3A4 with secondary contribution from monoamine oxidase A (MAO-A)supported

    Primary metabolism by CYP2D6 and CYP3A4; secondary MAO-A. pharmaceutics17040411

  • Meshkat et al. (2025), in a systematic review of psilocybin pharmacokinetics (14 studies, 112 human participants), reported for psilocin: half-life 1.5–4 hours, Tmax 1.8–4 hours oral, bioavailability 52.7 ± 20%, and primary metabolites 4-hydroxyindole-3-acetic acid (4-HIAA) and 4-hydroxytryptopholsupported

    Systematic review of 14 psilocybin PK studies (8 lab, 6 clinical; 112 healthy humans). Psilocin Tmax 1.8–4h; bioavailability 52.7±20%. Elimination half-life 1.5–4h. Metabolites: 4-HIAA and 4-hydroxytryptophol. pharmaceutics17040411

  • 4-AcO-DET is a presumed prodrug that undergoes hydrolysis via serum esterases to yield 4-HO-DET (ethocin/CZ-74), the pharmacologically active speciessupported

    Screened 28 novel psilocin prodrugs using 4-position cleavable groups including acetoxy. Novel prodrugs showed reduced pharmacological exposure vs psilocybin while retaining psilocin activity. acs.jmedchem.3c01225

  • Raithatha et al. (2024) demonstrated that 4-acetoxy groups on indole tryptamines function as cleavable prodrug moieties generating the active 4-hydroxy form, but noted that 4-acetoxy compounds show "somewhat weaker binding affinities but similar target profiles across serotonin receptors" compared to their 4-hydroxy counterparts.partially supported

    Novel prodrugs showed reduced pharmacological exposure vs psilocybin while retaining psilocin activity. acs.jmedchem.3c01225

  • Class-level data establish that 4-substituted tryptamines exhibit nanomolar 5-ht2a affinity with 25–380-fold selectivity over 5-ht1a and greater selectivity over 5-ht2c.partially supported

    4-hydroxylated derivatives show 25-380-fold selectivity for 5-HT2A vs 5-HT1A. N,N-dialkyl size secondary determinant. pmid:2139186

  • Class-level data establish that 4-substituted tryptamines exhibit nanomolar 5-ht2a affinity with 25–380-fold selectivity over 5-ht1a and greater selectivity over 5-ht2c.supported

    21 substituted tryptamines at 5-HT1A, 5-HT2A, 5-HT2C and SERT. 4-substituted compounds show high selectivity for 5-HT2AR. jpet.122.001454

  • Raithatha et al. (2024) found that 4-acetoxy prodrugs demonstrated "reduced pharmacological exposure compared with psilocybin," suggesting slower or less complete activation than phosphate ester prodrugs.supported

    Novel prodrugs showed reduced pharmacological exposure vs psilocybin while retaining psilocin activity. acs.jmedchem.3c01225

  • By analogy to Psilocin (4-HO-dmt), the active metabolite 4-HO-DET is expected to be metabolized primarily by CYP2D6 and CYP3A4 with secondary contribution from monoamine oxidase A (MAO-A)supported

    Primary metabolism by CYP2D6 and CYP3A4; secondary MAO-A. pharmaceutics17040411

  • Meshkat et al. (2025), in a systematic review of psilocybin pharmacokinetics (14 studies, 112 human participants), reported for psilocin: half-life 1.5–4 hours, Tmax 1.8–4 hours oral, bioavailability 52.7 ± 20%, and primary metabolites 4-hydroxyindole-3-acetic acid (4-HIAA) and 4-hydroxytryptopholsupported

    Systematic review of 14 psilocybin PK studies (8 lab, 6 clinical; 112 healthy humans). Psilocin Tmax 1.8–4h; bioavailability 52.7±20%. Elimination half-life 1.5–4h. Metabolites: 4-HIAA and 4-hydroxytryptophol. pharmaceutics17040411

comparative-pharmacology8 · 6 ok
  • Glatfelter et al. (2023) tested both N,N-diethyl and N,N-dimethyl 4-substituted tryptamines and found similar target profiles across serotonin receptors, though specific Ki values for 4-AcO-DET were not reported. 4-AcO-DMT has more extensive pharmacological characterization, including demonstrated nanomolar affinity for 5-ht2a and 5-ht1a.supported

    4-Acetoxy and 4-propionoxy analogues of 4-hydroxy compounds displayed somewhat weaker binding affinities but similar target profiles across 5-HT receptors and other identified targets. acsptsci.2c00222

  • Glatfelter et al. (2023) tested both N,N-diethyl and N,N-dimethyl 4-substituted tryptamines and found similar target profiles across serotonin receptors, though specific Ki values for 4-AcO-DET were not reported. 4-AcO-DMT has more extensive pharmacological characterization, including demonstrated nanomolar affinity for 5-ht2a and 5-ht1a.supported

    4-Acetoxy and 4-hydroxy compounds showed similar pharmacological effects. Psilacetin (4-AcO-DMT) acts as prodrug for psilocin. acsptsci.2c00177

  • Raithatha et al. (2024) demonstrated that acetate ester prodrugs showed reduced pharmacological exposure compared with psilocybin, suggesting the phosphate ester is a more efficient delivery mechanism.supported

    Novel prodrugs showed reduced pharmacological exposure vs psilocybin while retaining psilocin activity. acs.jmedchem.3c01225

  • McKenna et al. (1990) established that N,N-dialkyl group size influences 5-HT₂A binding affinitysupported

    N,N-dialkyl size secondary determinant; groups larger than diisopropyl markedly reduce affinity. pmid:2139186

  • Claim in comparative-pharmacology — inconclusiveinconclusiveworking →

  • Claim in comparative-pharmacology — inconclusiveinconclusiveworking →

  • Raithatha et al. (2024) demonstrated that acetate ester prodrugs showed reduced pharmacological exposure compared with psilocybin, suggesting the phosphate ester is a more efficient delivery mechanism.supported

    Novel prodrugs showed reduced pharmacological exposure vs psilocybin while retaining psilocin activity. acs.jmedchem.3c01225

  • McKenna et al. (1990) established that N,N-dialkyl group size influences 5-HT₂A binding affinitysupported

    N,N-dialkyl size secondary determinant; groups larger than diisopropyl markedly reduce affinity. pmid:2139186

summary8 · 7 ok
  • Claim in summary — inconclusiveinconclusiveworking →

  • As a presumed prodrug, 4-AcO-DET undergoes hydrolysis via serum esterases to yield 4-HO-DET, which acts as a 5-HT₂A receptor agonist — the primary mechanism of tryptamine psychedelicssupported

    4-acetoxy substitution modulates metabolic processing and pharmacokinetic profile. Novel prodrugs showed reduced pharmacological exposure vs psilocybin while retaining psilocin activity. acs.jmedchem.3c01225

  • No binding affinity data specific to 4-AcO-DET have been published; class-level data for 4-substituted tryptamines indicate nanomolar 5-HT₂A affinity with 25–380-fold selectivity over 5-ht1asupported

    4-hydroxylated derivatives show 25-380-fold selectivity for 5-HT2A vs 5-HT1A. pmid:2139186

  • The compound is not considered habit-forming; rapid tolerance develops via 5-HT₂A downregulation in the frontal cortex, with full reset estimated at approximately 7 days — a pharmacological ceiling on compulsive usepartially supported

    Tolerance after single dose. DOI caused 5-HT2AR downregulation in frontal cortex. acschemneuro.2c00170

  • Its active metabolite CZ-74 (4-HO-DET) was employed in European psycholytic therapy during the 1960s under Hanscarl Leuner, but no formal pharmacological characterization of 4-AcO-DET itself has been publishedsupported

    Comprehensive review of psycholytic therapy 1949-1970s, led by Hanscarl Leuner in Germany. Used at 30 European clinical centers. Various substances including tryptamines employed in serial lower-dose sessions. fpsyt.2022.1020505

  • As a presumed prodrug, 4-AcO-DET undergoes hydrolysis via serum esterases to yield 4-HO-DET, which acts as a 5-HT₂A receptor agonist — the primary mechanism of tryptamine psychedelicssupported

    Screened 28 novel psilocin prodrugs using 4-position cleavable groups including acetoxy. 4-acetoxy substitution modulates metabolic processing and pharmacokinetic profile. acs.jmedchem.3c01225

  • No binding affinity data specific to 4-AcO-DET have been published; class-level data for 4-substituted tryptamines indicate nanomolar 5-HT₂A affinity with 25–380-fold selectivity over 5-ht1asupported

    4-hydroxylated derivatives show 25-380-fold selectivity for 5-HT2A vs 5-HT1A. pmid:2139186

  • The compound is not considered habit-forming; rapid tolerance develops via 5-HT₂A downregulation in the frontal cortex, with full reset estimated at approximately 7 days — a pharmacological ceiling on compulsive usepartially supported

    Tolerance after single dose. DOI caused 5-HT2AR downregulation in frontal cortex. acschemneuro.2c00170

chemistry6 · 6 ok
  • This acetoxy moiety renders 4-AcO-DET a structural ester prodrug: hydrolysis cleaves the acetate group to yield 4-ho-det (ethocin/CZ-74), the active 4-hydroxytryptamine.supported

    Screened 28 novel psilocin prodrugs using 4-position cleavable groups including acetoxy. 4-acetoxy substitution modulates metabolic processing and pharmacokinetic profile. acs.jmedchem.3c01225

  • The N,N-diethyl substitution on the terminal amine distinguishes it from 4-AcO-DMT (N,N-dimethyl) and 4-aco-mipt (N-methyl-N-isopropyl), placing it in the middle range of N,N-dialkyl bulk — smaller than N,N-diisopropyl, which retains substantial 5-ht2a affinity, but larger than the dimethyl group found in the psilocin series.supported

    N,N-dialkyl size secondary determinant; groups larger than diisopropyl markedly reduce affinity. pmid:2139186

  • NMR reference spectra for structurally related 4-acetoxy tryptamines from grey-market samples are available, providing analytical reference for forensic and harm-reduction identification.supported

    1H NMR spectra of 43 alleged psychedelic tryptamines from grey-market vendors including 4-acetoxy compounds. Provides analytical reference spectra for forensic identification. isomerdesign-5-supp

  • This acetoxy moiety renders 4-AcO-DET a structural ester prodrug: hydrolysis cleaves the acetate group to yield 4-ho-det (ethocin/CZ-74), the active 4-hydroxytryptamine.supported

    Screened 28 novel psilocin prodrugs using 4-position cleavable groups including acetoxy. Novel prodrugs showed reduced pharmacological exposure vs psilocybin while retaining psilocin activity. acs.jmedchem.3c01225

  • The N,N-diethyl substitution on the terminal amine distinguishes it from 4-AcO-DMT (N,N-dimethyl) and 4-aco-mipt (N-methyl-N-isopropyl), placing it in the middle range of N,N-dialkyl bulk — smaller than N,N-diisopropyl, which retains substantial 5-ht2a affinity, but larger than the dimethyl group found in the psilocin series.supported

    N,N-dialkyl size secondary determinant; groups larger than diisopropyl markedly reduce affinity. pmid:2139186

  • NMR reference spectra for structurally related 4-acetoxy tryptamines from grey-market samples are available, providing analytical reference for forensic and harm-reduction identification.supported

    1H NMR spectra of 43 alleged psychedelic tryptamines from grey-market vendors including 4-acetoxy compounds. Provides analytical reference spectra for forensic identification. isomerdesign-5-supp

pharmacokinetic-detail6 · 6 ok
  • All parameters below are inferred by structural analogy to psilocin (4-HO-dmt), the best-characterized 4-hydroxytryptamine, via the systematic review of Meshkat et al. (2025).supported

    Systematic review of 14 psilocybin PK studies (8 lab, 6 clinical; 112 healthy humans). Psilocin Tmax 1.8–4h; Cmax 8.2±2.8 to 871 ng/mL (dose-dependent); bioavailability 52.7±20%. pharmaceutics17040411

  • Raithatha et al. (2024) found that 4-acetoxy prodrugs showed reduced pharmacological exposure compared to psilocybin (a phosphate ester prodrug), suggesting either slower hydrolysis, incomplete conversion, or different tissue distributionsupported

    Novel prodrugs showed reduced pharmacological exposure vs psilocybin while retaining psilocin activity. acs.jmedchem.3c01225

  • cyp2d6 is the primary metabolic enzyme for psilocinpartially supported

    Primary metabolism by CYP2D6 and CYP3A4; secondary MAO-A. pharmaceutics17040411

  • All parameters below are inferred by structural analogy to psilocin (4-HO-dmt), the best-characterized 4-hydroxytryptamine, via the systematic review of Meshkat et al. (2025).supported

    Systematic review of 14 psilocybin PK studies (8 lab, 6 clinical; 112 healthy humans). pharmaceutics17040411

  • Raithatha et al. (2024) found that 4-acetoxy prodrugs showed reduced pharmacological exposure compared to psilocybin (a phosphate ester prodrug), suggesting either slower hydrolysis, incomplete conversion, or different tissue distributionsupported

    Novel prodrugs showed reduced pharmacological exposure vs psilocybin while retaining psilocin activity. acs.jmedchem.3c01225

  • cyp2d6 is the primary metabolic enzyme for psilocinpartially supported

    Primary metabolism by CYP2D6 and CYP3A4; secondary MAO-A. pharmaceutics17040411

history4 · 4 ok
  • CZ-74 entered human clinical use during the 1960s within the framework of psycholytic therapy — the European paradigm of psychedelic-assisted psychotherapy. Hanscarl Leuner of the University of Göttingen, the leading figure in European psycholytic therapy and head of the European Medical Society for Psycholytic Therapy (ept), employed CZ-74 and CEY-19 across approximately 30 clinical treatment centers in Europesupported

    Comprehensive review of psycholytic therapy 1949-1970s, led by Hanscarl Leuner in Germany. Used at 30 European clinical centers. Various substances including tryptamines employed in serial lower-dose sessions. fpsyt.2022.1020505

  • The broader psycholytic therapy era from which CZ-74 emerged is reviewed by Passie et al. (2022).supported

    Comprehensive review of psycholytic therapy 1949-1970s, led by Hanscarl Leuner in Germany. fpsyt.2022.1020505

  • CZ-74 entered human clinical use during the 1960s within the framework of psycholytic therapy — the European paradigm of psychedelic-assisted psychotherapy. Hanscarl Leuner of the University of Göttingen, the leading figure in European psycholytic therapy and head of the European Medical Society for Psycholytic Therapy (ept), employed CZ-74 and CEY-19 across approximately 30 clinical treatment centers in Europesupported

    Comprehensive review of psycholytic therapy 1949-1970s, led by Hanscarl Leuner in Germany. Used at 30 European clinical centers. Various substances including tryptamines employed in serial lower-dose sessions. fpsyt.2022.1020505

  • The broader psycholytic therapy era from which CZ-74 emerged is reviewed by Passie et al. (2022).supported

    Comprehensive review of psycholytic therapy 1949-1970s, led by Hanscarl Leuner in Germany. fpsyt.2022.1020505

neuroscience4 · 3 ok
  • Cameron et al. (2023) reviewed evidence that serotonergic psychedelics interact with TrkB neurotrophin receptors, promoting structural neuroplasticity via bdnf/mTOR signaling cascadessupported

    Review of psychedelic mechanisms beyond 5-HT2A including 5-HT1A, 5-HT2C, TrkB receptors. Neuroplasticity promotion reviewed. jneurosci.1384-23.2023

  • Claim in neuroscience — inconclusiveinconclusiveworking →

  • Cameron et al. (2023) reviewed evidence that serotonergic psychedelics interact with TrkB neurotrophin receptors, promoting structural neuroplasticity via bdnf/mTOR signaling cascadessupported

    Review of psychedelic mechanisms beyond 5-HT2A including 5-HT1A, 5-HT2C, TrkB receptors. Neuroplasticity promotion reviewed. jneurosci.1384-23.2023

  • The psychoplastogen hypothesis proposes that psychedelics promote rapid structural neuroplasticity — dendritic arbor growth, spinogenesis, and synaptogenesis — via 5-HT₂A/TrkB/BDNF/mTOR signalingsupported

    Review of psychedelic mechanisms beyond 5-HT2A including 5-HT1A, 5-HT2C, TrkB receptors. Neuroplasticity promotion reviewed. jneurosci.1384-23.2023

clinical-evidence2 · 2 ok
  • The active metabolite CZ-74 was employed in clinical psycholytic therapy during the 1960s by Hanscarl Leuner and colleagues at approximately 30 European treatment centerssupported

    Comprehensive review of psycholytic therapy 1949-1970s, led by Hanscarl Leuner in Germany. Used at 30 European clinical centers. Various substances including tryptamines employed in serial lower-dose sessions. fpsyt.2022.1020505

  • The active metabolite CZ-74 was employed in clinical psycholytic therapy during the 1960s by Hanscarl Leuner and colleagues at approximately 30 European treatment centerssupported

    Comprehensive review of psycholytic therapy 1949-1970s, led by Hanscarl Leuner in Germany. Used at 30 European clinical centers. Various substances including tryptamines employed in serial lower-dose sessions. fpsyt.2022.1020505

effects-overview2 · 1 ok
  • Claim in effects-overview — inconclusiveinconclusiveworking →

  • No controlled human studies have documented its subjective effects; all characterization derives from clinical reports on its active metabolite CZ-74 (4-ho-det) and community experience reports.supported

    Various substances including tryptamines employed in serial lower-dose sessions. fpsyt.2022.1020505

3Sources

25 consumed · sorted by year · feeds = sections resting on it

Sources
yearsourcetierstatusfeeds
2025Pharmacokinetics of Psilocybin: A Systematic ReviewMeshkat S, Al-Shamali H, Perivolaris A, et al. · Pharmaceuticsdoi:10.3390/pharmaceutics17040411strongconsumed3 sections
20244-Acetoxy-DET (Ethacetin) - WikidocWikidoc contributorsurl:a9a7a4460a42dd90weakconsumedcited only
20244-AcO-DET (Ethacetin) compound recordPubChemurl:47d86a78d170c66bmoderateconsumedcited only
2024Novel Psilocin Prodrugs with Altered Pharmacological Properties as Candidate Therapies for Treatment-Resistant Anxiety DisordersRaithatha SA, Hagel JM, Matinkhoo K, Yu L, Press D, Cook SG, et al. · Journal of Medicinal Chemistrydoi:10.1021/acs.jmedchem.3c01225moderateconsumed5 sections
20234-HO-DET (CZ-74) — DMT-Nexus WikiDMT-Nexus contributorsurl:b18ca63773199877weakconsumedcited only
2023Beyond the 5-HT2A Receptor: Classic and Nonclassic Targets in Psychedelic Drug ActionCameron LP, Benetatos J, Lewis V, Bonniwell EM, Jaster AM, et al. · Journal of Neurosciencedoi:10.1523/jneurosci.1384-23.2023moderateconsumed2 sections
2023Pharmacologic Activity of Substituted Tryptamines at 5-HT2A, 5-HT2C, 5-HT1A Receptors and SERTKozell LB, Eshleman AJ, Swanson TL, Bloom SH, Wolfrum KM, et al. · Journal of Pharmacology and Experimental Therapeuticsdoi:10.1124/jpet.122.001454moderateconsumed2 sections
2023Receptor Binding Profiles for Tryptamine Psychedelics and Effects of 4-Propionoxy-N,N-dimethyltryptamine in MiceGlatfelter GC, Naeem M, Pham DNK, Golen JA, Chadeayne AR, Manke DR, Baumann MH · ACS Pharmacology & Translational Sciencedoi:10.1021/acsptsci.2c00222strongconsumed2 sections
2022Lower-Dose Psycholytic Therapy - A Neglected ApproachPassie T, Guss J, Krähenmann R · Frontiers in Psychiatrydoi:10.3389/fpsyt.2022.1020505moderateconsumed4 sections
2022Marked and prolonged serotonin toxicity in a tramadol-poisoned patient with a pharmacokinetic studyBianconi G, Malissin I, Labat L, Khoudour N, Houzé P, Pallet N, Mégarbane B, Declèves X · Clinical Toxicologydoi:10.1080/15563650.2021.1955912weakconsumed1 section
2022Schedules of Controlled Substances: Proposed Placement of 4-OH-DiPT, 5-MeO-AMT, 5-MeO-MiPT, 5-MeO-DET, and DiPT in Schedule IDEA / Department of Justiceurl:bf5b46070f6f77a2weakconsumedcited only
2022Structure-Activity Relationships for Psilocybin, Baeocystin, Aeruginascin, and Related Analogues to Produce Pharmacological Effects in MiceGlatfelter GC, Pottie E, Partilla JS, Sherwood AM, et al. · ACS Pharmacology and Translational Sciencedoi:10.1021/acsptsci.2c00177moderateconsumed2 sections
2022Tolerance and Cross-Tolerance among Psychedelic and Nonpsychedelic 5-HT2A Receptor Agonists in Micede la Fuente Revenga M, Jaster AM, McGinn J, Silva G, Saha S, Gonzalez-Maeso J · ACS Chemical Neurosciencedoi:10.1021/acschemneuro.2c00170moderateconsumed2 sections
2020Correlation between the potency of hallucinogens in the mouse head-twitch response assay and their behavioral and subjective effects in other speciesHalberstadt AL, Chatha M, Klein AK, Wallach J, Brandt SD · Neuropharmacologydoi:10.1016/j.neuropharm.2019.107933moderateconsumed1 section
2020Toxicology and Analysis of Psychoactive Tryptamines.Malaca S, Lo Faro AF, Tamborra A, Pichini S, Busardò FP, Huestis MA · International journal of molecular sciencesdoi:10.3390/ijms21239279moderateconsumed1 section
2018Opioid analgesic drugs and serotonin toxicity (syndrome): mechanisms, animal models, and links to clinical effectsBaldo BA · Archives of Toxicologydoi:10.1007/s00204-018-2244-6moderateconsumed1 section
2017PeakAL: Protons I Have Known and Loved, Too - Another Fifty Shades of Grey-Market Spectra. Supplementary DataChapman SJ · IsomerDesign Technical Reportdoi:10.16889/isomerdesign-5-suppmoderateconsumed1 section
2016Receptor interaction profiles of novel psychoactive tryptamines compared with classic hallucinogensRickli A, Moning OD, Hoener MC, Liechti ME · European Neuropsychopharmacologydoi:10.1016/j.euroneuro.2016.05.001strongconsumed2 sections
2015Hanscarl Leuner: Pioneer of Hallucinogen Research and Psycholytic TherapyMAPSurl:c0d1fa9c049ba571weakconsumedcited only
2015Recreational use, analysis and toxicity of tryptamines.Tittarelli R, Mannocchi G, Pantano F, Romolo FS · Current neuropharmacologydoi:10.2174/1570159x13666141210222409moderateconsumedcited only
2012Avoiding serotonin syndrome: the nature of the interaction between tramadol and selective serotonin reuptake inhibitorsNelson EM, Philbrick AM · Annals of Pharmacotherapydoi:10.1345/aph.1q748moderateconsumedcited only
2011Abuse liability profile of three substituted tryptaminesGatch MB, Forster MJ, Janowsky A, Eshleman AJ · Journal of Pharmacology and Experimental Therapeuticsdoi:10.1124/jpet.111.179705strongconsumed1 section
20014-AcO-DET/Ethacetin Degradation (Erowid)Erowidurl:4cc1e24bda32b779weakconsumedcited only
1990Differential interactions of indolealkylamines with 5-hydroxytryptamine receptor subtypesMcKenna DJ, Repke DB, Lo L, Peroutka SJ · Neuropharmacologypmid:2139186moderateconsumed5 sections
1973DEA Scheduling: PMA as Schedule I hallucinogen (38 FR 26447, July 2, 1973)US Drug Enforcement Administrationurl:c843cbe335da96c8weakconsumedcited only
—Searched ClinicalTrials.gov for "4-AcO-DET ethacetin" — no resultsabsenceno results—
—Searched OpenAlex for "4-acetoxy-DET ethacetin tryptamine psychedelic" — no resultsabsenceno results—
—Searched PubMed for "4-AcO-DET ethacetin diethyltryptamine acetate pharmacology" — no resultsabsenceno results—

4Docket

one filing per activity — decisions, correspondence, reports

  1. 0012026-04-28revisionRevision 1

    Initial LLM generation (v5 pipeline, wiki)

    psx:substance/4-aco-det/version/1
  2. 0022026-06-04proposal28 value proposals approved together

    one review action inferred from a shared reviewer and instant — the graph records one activity per proposal

    28 items in this filing

    • dose_tier · light
    • dose_tier · common
    • dose_tier · strong
    • dose_tier · heavy
    • timeline · oral · onset
    • timeline · oral · come_up
    • timeline · oral · peak
    • timeline · oral · resolution
    • timeline · oral · afterglow
    • toxicity
    • dependence · full
    • withdrawal
    • tolerance
    • dosage · oral
    • duration · oral · onset
    • duration · oral · come_up
    • duration · oral · peak
    • duration · oral · offset
    • duration · oral · total
    • duration · oral · aftereffects
    • contraindication · psychiatric
    • contraindication · psychiatric
    • contraindication · other
    • contraindication · other
    • contraindication · cardiovascular
    • contraindication · pregnancy_breastfeeding
    • contraindication · other
    psx:substance/4-aco-det/proposal/dose_tier/e148ccc5-7461-4c24-bcdc-9df81ea1b364
  3. 0032026-06-04publicationPublication — backfill

5Issued documents

what the notice, info card and emergency page said, by hash

Issued documents
documentcontent hashissuedfrom versionsuperseded
emergencyc15c760aabf6f0fcf5b6fc6b…2026-10-19 20:29Revision 1current
info_card6384cb4788288ce16027c3d0…2026-10-19 20:29Revision 1current
noticef069a9dea47b308467344618…2026-10-19 20:29Revision 1current
emergency3f0909c25c714d225b4bb3e3…2026-09-04 04:27Revision 1superseded2026-10-19
info_carde287cd50add82c29980418d7…2026-09-04 04:27Revision 1superseded2026-10-19
notice64878b43a1d2932a5305f55b…2026-09-04 04:27Revision 1superseded2026-10-19