Self-hypnosis is a trance practice rooted in the secular clinical hypnosis tradition, developed as a standalone skill from James Braid's 1843 self-experiments. It is the most extensively studied self-administered hypnotic technique, supported by 22 randomized controlled trials across pain, anxiety, and procedural medicine.[1] It alters consciousness primarily through self-directed suggestion — a focused, absorbed attentional state in which verbal or imagined cues shape experience.
The practitioner adopts a comfortable seated or reclining posture and begins with an induction — eye fixation, progressive muscle relaxation, or counting down — that narrows attention into a focused, absorbed state. Once that state is established, the practitioner delivers a pre-prepared suggestion targeting a specific goal: pain reduction, sleep onset, habit modification, or symptom relief. Sessions last 10 to 30 minutes and are typically practiced daily or as needed.
A typical session lasts 10 to 30 minutes. At introductory levels, practitioners report deep physical relaxation, narrowed attention, a sense of mental quiet, and mild time distortion. With practice, the absorbed state deepens and suggestions act more reliably on target symptoms — pain, anxiety, sleep, or bowel function. Boredom, distraction, mild headache, intrusive thoughts, and occasional emotional surfacing are common; depth and effect size are partly moderated by trait hypnotizability.[2]
Risks are low but real: transient headache, dizziness, and unexpected emotional surfacing are the most common adverse effects, and the practice can amplify dissociative symptoms in vulnerable individuals.[3] Untreated psychosis, severe dissociative disorder, borderline personality disorder, and active suicidal ideation are contraindications for unsupervised practice. Self-hypnosis remains one of the few self-administered psychological interventions with a substantial clinical evidence base, embedded in pain medicine, IBS care, and procedural anesthesia worldwide.
History & Tradition
Origins
The modern concept of self-hypnosis traces to the work of the Scottish surgeon James Braid (1795–1860), who coined the term "hypnotism" in 1841 after observing that sustained eye-fixation produced a state of attentional absorption independent of any "animal magnetism" attributed to the operator. On 1 May 1843, Braid hypnotized himself — the first documented systematic autohypnosis — having already taught the technique to clients.[4] His self-experiments demonstrated that the hypnotic state resided in the subject's own attentional process rather than in any quality of the operator, setting the conceptual stage for self-hypnosis as a standalone skill.
The French pharmacist Émile Coué (1857–1926) developed the doctrine of "conscious autosuggestion" in the early 20th century. Coué observed that the therapeutic effect of medications correlated with how they were described to patients, and abstracted this into a self-directed verbal formula — most famously "every day, in every way, I am getting better and better." His clinic at Nancy attracted international attention, and his method was disseminated widely as lay self-help. Coué's emphasis on imagination over willpower prefigured later sociocognitive accounts of hypnotic response.
The German psychiatrist Johannes Schultz (1884–1970) systematized autogenic training in 1932 by formalizing the self-directed heaviness and warmth sensations his hypnotized patients spontaneously reported. Schultz drew directly on the hypnosis work of Oskar Vogt and aimed to give patients a self-administered physiological relaxation technique without requiring a clinician present. Autogenic training is a parallel derivation that shares the autosuggestion mechanism but is not synonymous with self-hypnosis — it is a distinct lineage with its own German clinical literature.
Transmission & secularization
The American hypnotist Dave Elman (1900–1967) codified rapid-induction techniques that became foundational to mid-20th-century clinical hypnotherapy pedagogy. His 1964 text Hypnotherapy included a dedicated self-hypnosis chapter and described the fractionation technique — alternating light and deep trance states — which Elman considered the most efficient pathway to profound trance.[5]
From the 1960s onward, self-hypnosis shifted progressively into clinical institutional contexts. The American Society of Clinical Hypnosis (ASCH), founded in 1957, and the Society for Clinical and Experimental Hypnosis (SCEH) established training curricula and ethical frameworks. The British Society of Clinical and Academic Hypnosis (BSCAH) occupies an equivalent role in the United Kingdom. These bodies standardized self-hypnosis as a between-session skill taught to patients by a credentialed clinician, rather than as a freestanding tradition with its own teachers and lineage holders.
Contemporary context
Two distinct contexts now exist in parallel. The clinical context is governed by professional credentialing bodies (ASCH, SCEH, BSCAH), which restrict certification to licensed healthcare practitioners and treat self-hypnosis as one component of clinical hypnotherapy.[6] The consumer context — recorded audio inductions, smartphone applications, lay courses — operates largely outside these frameworks. The Elkins 2018 systematic review found that the method of delivery matters: interactive instruction by a trained practitioner, with at least three self-practice sessions before measurement, produced reliable effects, whereas passive listening to audio recordings of heterohypnosis often did not.[1] Active research continues, including a registered clinical trial on self-hypnosis training for chronic pain management.[7]
The Practice
Technique
The practitioner adopts a comfortable, supported posture — seated upright in a chair with feet flat and hands resting on the thighs, or reclining on a couch or bed with the body fully supported. The essential requirement is a position that can be maintained without postural effort for the duration of the session, since muscular work competes with the absorption the induction aims to produce. The environment is quiet and free from interruption; eyes are typically closed, though eye-fixation inductions begin with the gaze fixed on a small visual target.
The method proceeds in three components: induction, deepening, and suggestion. Induction is the procedure that shifts attention from broad ambient awareness into a focused, absorbed state — common variants include eye-fixation (sustained gaze on a single point until the eyelids feel heavy and close), progressive muscular relaxation (systematically tensing and releasing each muscle group), and counting down (counting backward from a chosen number while pairing each count with a phrase such as "deeper and more relaxed"). Once induction is achieved, the practitioner deepens the state — typically by imagining a descending staircase, repeated relaxation cues, or Elman's fractionation method of briefly opening the eyes and re-inducing to rebound into a deeper level.[5] The practitioner then delivers one or two pre-prepared suggestions — short, positive, present-tense statements targeting a specific outcome (e.g., "my hand is cool and comfortable" for pain management). The session closes with a counted exit returning to ordinary alertness. The most common departure from the method is treating relaxation as the goal rather than as a precondition: the absorbed, suggestible state is what makes the suggestion act, and relaxation without suggestion produces only relaxation.
At the introductory level, sessions run 10–15 minutes and contain a single, well-prepared suggestion; the practitioner is still learning to recognize the shift in attentional quality that marks successful induction. At the established level, sessions extend to 15–30 minutes with a tailored deepening protocol and suggestions developed for a defined therapeutic goal; clinical guidance holds that at least three self-practice sessions are needed before a suggestion produces measurable effect, a threshold consistent with the Elkins meta-analytic methodology finding.[1] With extended practice, anchors and cues can be conditioned: a specific word, gesture, or breath pattern can reliably re-enter the absorbed state within seconds, enabling 2–5 minute micro-sessions with effects comparable to full-length sittings.
Two principal variants share the underlying mechanism but differ in induction and emphasis. Coué-style autosuggestion centers on the verbal formula repeated in a lightly absorbed state, without elaborate induction or deepening. Schultz's autogenic training is a distinct German clinical derivation that uses six standard self-statements ("my right arm is heavy," "my right arm is warm," and so on) practiced over months to produce a reliable physiological relaxation response; it overlaps with self-hypnosis at the mechanism level but is taught and practiced as its own technique. Gut-directed hypnotherapy for irritable bowel syndrome, codified in standardized clinical protocols, is a content-specific variant in which patients learn self-hypnosis with suggestions targeted to bowel function.[8]
Setting & Preparation
The practice requires no equipment beyond a quiet, interruption-free space and a supported posture. A timer set to silence after the planned session duration is useful to remove the cognitive load of clock-watching. Self-hypnosis is normally practiced alone; group practice is uncommon outside of teaching contexts. A trained clinician is required for the initial skill acquisition phase when the practice is being used for a clinical indication — the Elkins meta-analysis found that interactive instruction outperformed passive audio delivery.[1] Audio recordings and smartphone applications are widely used for self-administration once the skill is established, though evidence for audio-only delivery without prior practitioner instruction is meaningfully weaker.
Practice in a quiet, interruption-free space. A reclining chair, couch, or bed with full body support reduces the postural effort that would otherwise compete with absorption. Set a silent timer for the planned session length so clock-watching does not intrude on attention. For the first weeks of practice, schedule sessions at consistent times — early morning or just before sleep are common — to build the conditioned association between the setting and the state.
For clinical applications (pain, IBS, anxiety, hot flashes, procedural preparation), the strongest evidence supports an initial period of interactive instruction with a credentialed practitioner before transitioning to independent self-practice.[1] Audio recordings can support established practice but are a weaker substitute for skill acquisition.
Guidelines
- Do not attempt unsupervised self-hypnosis if you have untreated psychosis, severe dissociative disorder, borderline personality disorder, active suicidal ideation, dementia, or significant cognitive impairment.
- Do not practice while intoxicated by alcohol, benzodiazepines, opioids, or other CNS depressants.
- Start with introductory-tier sessions of 10–15 minutes containing a single, pre-prepared suggestion; expect three or more practice sessions before measurable effects emerge.
- Do not use self-hypnosis for memory exploration or trauma work — the risk of generating false memory is significant and clinical supervision is required for this purpose.
- Do not substitute self-hypnosis for medical diagnosis of a somatic symptom; use it as an adjunct to, not in place of, medical evaluation.
- If emotional content surfaces unexpectedly and persists past the session, end the practice for that period and seek clinical support before resuming.
- For clinical indications, learn the skill from a credentialed practitioner (ASCH, SCEH, BSCAH) before transitioning to independent home practice.
Mechanism
Attentional absorption and salience network changes
The most replicated neural signature of the hypnotic state involves the salience network and the executive control network. In a Stanford fMRI study of 57 subjects (36 high-hypnotizable, 21 low), hypnosis in high hypnotizables produced three concurrent changes: decreased activity in the dorsal anterior cingulate cortex (dACC), a node of the salience network; increased functional connectivity between the dorsolateral prefrontal cortex (DLPFC) and the insula, interpreted as enhanced brain–body regulatory coupling; and reduced connectivity between the DLPFC and the default mode network (DMN), interpreted as a dissociation between executed action and self-referential awareness.[2] These changes were absent in low hypnotizables under the same conditions, indicating a trait-dependent neural mechanism rather than a generic relaxation response. EEG studies corroborate the picture: hypnotic induction in high-susceptible individuals produces state-like changes in theta and beta band functional connectivity consistent with reduced goal-directed monitoring and increased inward attentional focus.[9]
Suggestion as cognitive-control re-routing
The therapeutic action of self-hypnosis is mediated by the suggestion, not by the trance per se. The proposed mechanism is that the absorbed state suspends the ordinary critical evaluation that would otherwise reject a self-administered instruction, permitting the suggestion to act on perception, affect, or autonomic output. The increased DLPFC–insula coupling observed during hypnosis in high hypnotizables provides a candidate neural substrate: the executive control network gains direct regulatory access to interoceptive and autonomic systems normally outside voluntary control.[2] This account is consistent with the observed pattern of clinical effects — pain modulation, autonomic shifts, smooth-muscle effects in IBS — and with the finding that effect size scales with hypnotizability and with active practice rather than with relaxation alone.
Hypnotizability as moderator
Trait hypnotizability — the stable individual capacity for hypnotic response — is normally distributed in the general population, with roughly 10–15% of adults being high responders on the Stanford Hypnotic Susceptibility Scale and a similar fraction being low responders.[10] A 2024 review of resting-state fMRI and EEG concluded that hypnotizability is a neurobiologically grounded stable trait, with characteristic salience- and default-mode-network connectivity differences observable outside hypnotic states.[11] The Stanford SHIFT trial integrated hypnotizability-targeted transcranial stimulation as an adjunct, indicating active translational research connecting this neuroscience to clinical enhancement.[12]
Physiological correlates
Independent of neuroimaging, hypnosis is associated with reductions in cortisol, heart-rate variability shifts consistent with parasympathetic activation, reduced skin conductance responses to noxious stimuli, and altered pain tolerance on cold-pressor tasks. Effect magnitudes scale with trait hypnotizability and with active engagement: motivated self-practice produces larger physiological changes than passive listening to recordings.
Several mechanistic puzzles remain unresolved in the self-hypnosis literature.
Subjective Effects
Self-hypnosis produces a phenomenologically distinctive altered state characterized by focused absorption, narrowed attentional field, deep physical relaxation, mild time distortion, and heightened responsiveness to verbal or imagined suggestion. Subjective depth ranges along a continuum from light absorption — barely distinguishable from relaxed wakefulness — through medium trance to deep trance with vivid imagery and altered body sense. Depth is moderated by trait hypnotizability and by accumulated practice rather than by elapsed clock time within a session.
At the introductory level, effects are dominated by physical relaxation, mental quiet, and a felt sense of attentional narrowing onto the induction anchor. Mild time distortion (sessions feeling shorter or longer than they are) is common. At the established level, with reliable self-induction in place, practitioners report clearer access to a felt shift in state, vivid imagery in response to suggestion, reduced reactivity to ambient stimuli, and the emergence of conditioned anchors that re-enter the state on cue. Suggestion-targeted effects — analgesia, reduced anxiety, autonomic shifts, sleep onset — typically become measurable after three or more practice sessions.[1]
Unsought effects include boredom, restlessness, intrusive thoughts that compete with the suggestion, mild headache or dizziness on exit, and occasional emotional surfacing — recollection of affect-laden material that the practitioner did not intend to access. Effect depth is partly determined by trait hypnotizability: approximately 10–15% of adults score as high hypnotizables on standardized scales and access the deepest states most readily,[10] while low hypnotizables may achieve relaxation through self-hypnosis without accessing deeper trance phenomena.
▸▾Effect Grid
Induction (relaxation + focus) and a single suggestion.
Safety
Adverse Effects
Self-hypnosis has a favorable safety profile relative to most clinical psychological interventions. No serious adverse events have been documented across the 49 meta-analyses reviewed by Rosendahl and colleagues in their 20-year evidence synthesis.[13] A BSCAH review of unwanted effects categorizes adverse experiences into two clusters — emotional and physical — and reports that both are typically transient and mild.[3]
Physical adverse effects
The most frequently reported physical adverse effects are transient headache, dizziness, nausea, and sweating, typically resolving within minutes to hours after session end. These are reported across clinical trials at low frequencies and rarely require intervention.
Emotional adverse effects
The emotionally significant adverse effects are the unintended retrieval of affect-laden memory or the surfacing of distressing affect during a session targeted at an unrelated goal. Hypnosis can facilitate vivid retrieval of biographical material, and without a clinician present this content can be disorienting.[3] This risk is proportionally higher in self-hypnosis than in clinician-administered hypnosis because there is no external container for distressing material.
A specific and clinically significant risk is the inadvertent generation of false memory when self-hypnosis is used for memory exploration or trauma work. Hypnotic states heighten suggestibility and imaginative elaboration, and self-generated suggestions during hypnosis can produce conviction in reconstructed rather than recalled content. Use of hypnosis for forensic or trauma memory purposes is discouraged across professional bodies and is outside the scope of self-practice.
Symptom masking is a further concern: using self-hypnosis for pain management without medical diagnosis risks suppressing a symptom that requires investigation.
Contraindications
Psychiatric contraindications
Unsupervised self-hypnosis is contraindicated in several psychiatric conditions. Untreated psychosis or schizophrenia is a contraindication because hypnotic states can further blur reality testing. Severe dissociative disorder is a contraindication because self-hypnosis is itself a dissociation-adjacent state and can amplify pathological dissociation rather than producing focused absorption.[3] Borderline personality disorder is a relative contraindication for unsupervised practice because hypnosis can intensify emotional lability. Active suicidal ideation is a contraindication because the practice should not be substituted for crisis-level clinical care.
Medical and situational contraindications
Dementia or significant cognitive impairment is a contraindication because reliable self-induction and self-exit require intact executive function. Drug or alcohol intoxication is a contraindication because central nervous system depressants compound dissociative depth and impair voluntary exit from the state. Trauma history characterized by pathological dissociation is a relative contraindication: such practitioners may find that self-hypnosis amplifies dissociative symptoms rather than producing the relaxed absorbed state, and professional supervision is recommended for initial skill acquisition.
Substance Interactions
No dedicated pharmacological interaction studies between self-hypnosis and psychoactive substances have been published. The considerations below are mechanistic rather than empirically tested, and absence of controlled data should itself be treated as a finding.
Central nervous system depressants — alcohol, benzodiazepines, opioids — deepen sedation and reduce voluntary cognitive control. Self-hypnosis conducted under their influence may produce unpredictable dissociative depth or impair the capacity to exit the state voluntarily; no formal studies characterize this interaction. Stimulant-mediated sympathetic arousal may impair induction and deepening, given that hypnotic induction is facilitated by parasympathetic tone; empirical evidence is absent. Psychedelics and dissociatives share phenomenological features with hypnotic states (altered agency, absorption, altered temporality); combining self-hypnosis with these substances is not studied and raises theoretical concerns about compounding reality-testing alterations in individuals with predisposing vulnerabilities.
A separate clinical literature on adjunctive hypnosis in surgical contexts documents reduced anesthetic requirements (g=0.38 for medication consumption across 34 RCTs in adult surgical patients).[14] This confirms that hypnotic states interact with drug dose-response in clinically meaningful ways in controlled settings, but does not directly inform recreational or habit-use combinations.
Habituation
Self-hypnosis carries low habituation and compulsive-use risk. The practice has not been associated with a recognized dependence syndrome in the clinical literature, and the Rosendahl 20-year review reports no documented serious adverse pattern consistent with compulsive use.[13] Practitioners may, however, develop psychological reliance on self-hypnosis as a substitute for medical evaluation — using the practice to suppress a symptom rather than to investigate its cause. This is a use-pattern concern rather than a pharmacological one, and is addressed by the standing guidance that self-hypnosis for somatic symptoms should follow rather than precede medical diagnosis.
If Distress Arises
A practitioner can exit self-hypnosis voluntarily at any point — this is the practice's principal safety margin compared with substance-induced altered states. The standard exit procedure is a slow count from one to five, paired with cues for returning alertness ("at five I will open my eyes, feeling refreshed and fully awake"). If practice is interrupted by external disturbance, the state typically resolves on its own within seconds to a minute.
If distressing emotional material surfaces during a session, the appropriate response is to end the session rather than push through. Use the counted exit, sit upright, drink water, and ground in the physical environment (name three objects you can see, three you can hear). If the affect persists or returns repeatedly across sessions, pause self-practice and consult a credentialed hypnotherapist or mental health clinician before resuming. If physical adverse effects (headache, dizziness, nausea) appear mid-session, end the session — these typically resolve within minutes to hours but are a signal to adjust induction style or session length on the next attempt.[3]
Recovery & Support
For practitioners encountering persistent emotional difficulty associated with self-hypnosis practice, the appropriate referral is to a clinician credentialed by a recognized professional body — ASCH or SCEH in the United States, BSCAH in the United Kingdom, the Australian Society of Hypnosis in Australia. These bodies restrict certification to licensed healthcare practitioners on the grounds that hypnosis competence presupposes diagnostic and therapeutic competencies beyond hypnosis technique alone.[6]
For acute psychiatric concerns — surfacing of trauma material, dissociative symptoms, suicidal ideation triggered or amplified by practice — standard crisis resources apply (national suicide and crisis lines, emergency department) and self-practice should be paused until clinical evaluation has occurred. Self-hypnosis carries no recognized dependence syndrome, so recovery-from-practice is not the typical concern; the recovery concern is access to appropriate clinical evaluation when practice produces unintended emotional or psychiatric effects.
Recovery resources, crisis lines, and support pathways → View all
Progression
Skill acquisition in self-hypnosis follows a roughly three-phase trajectory documented across clinical training literatures, though no single canonical stage map exists as it does for some contemplative traditions.
Induction competence
In the first phase, typically weeks 1–4, the practitioner is learning to reliably enter a focused, absorbed state using a chosen induction method — eye fixation, progressive muscle relaxation, counting down, or Elman fractionation. The criterion is consistent replicability of a subjective shift: a recognized change in attentional quality that the practitioner can produce on demand, not merely on lucky sessions. Daily 10–15 minute practice is typical at this phase.
Deepening and suggestion delivery
In the second phase, typically overlapping weeks 2–8, the practitioner develops a personalized deepening protocol and a specific suggestion set targeted to a therapeutic goal. Clinical guidance holds that at least three self-practice sessions are needed before suggestions produce measurable effects — a threshold consistent with the methodology finding from the Elkins systematic review that participants who completed three or more practice sessions showed reliable effects, whereas those receiving passive audio without practice often did not.[1]
Tailored and conditioned response
With extended practice, cues and anchors can be conditioned: a specific word, gesture, or sensory stimulus reliably and rapidly re-enters the state. Elman described this conditioned-response efficiency as fractionation's principal clinical value.[5] At this stage, a 2–5 minute micro-session can produce outcomes comparable to longer sessions, allowing the practice to be deployed in situ — before a medical procedure, during a flare of chronic pain, at the onset of insomnia.
Trait hypnotizability moderates the trajectory. High hypnotizables (approximately 10–15% of the population on the Stanford scale) show rapid acquisition and deep states; low hypnotizables may achieve relaxation and mild suggestion effects through self-hypnosis without accessing pronounced trance phenomena.[10] Applications targeting relaxation and mild suggestion are accessible across the hypnotizability range; applications requiring deep-trance phenomena (amnesia, positive hallucination) are disproportionately effective in high-hypnotizable individuals.
Evidence Summary
The self-hypnosis and clinical hypnosis evidence base has grown substantially over the past two decades and is now among the strongest for any self-administered psychological intervention.
Direct self-hypnosis evidence
The Elkins 2018 systematic review and meta-analysis is the most direct evidence base. Starting from 576 studies identified by searches on "self-hypnosis," "autohypnosis," "autosuggestion," and "self-hypnotic," the review retained 22 RCTs meeting inclusion criteria, with self-hypnosis showing efficacy across pain, childbirth, paediatric applications, stress, and anxiety.[1] A methodological finding from this review is consequential for practice: effectiveness depended on teaching self-hypnosis as an independent skill with at least three practice sessions before measurement, whereas trials using passive audio recordings of heterohypnosis typically showed weaker or null effects.
Broader hypnosis evidence
A 20-year meta-analytic review covering 49 meta-analyses across 261 distinct primary studies found Cohen's d ranging from −0.04 to 2.72; approximately 25.4% of effects were medium-sized (d ≥ 0.5) and 28.8% large (d ≥ 0.8).[13] The strongest evidence concerns patients undergoing medical procedures, pain patients, and children and adolescents. A meta-analysis of 34 RCTs (N=2,597) in adult surgical and procedural populations found positive effects on emotional distress (g=0.53), pain (g=0.44), and medication consumption (g=0.38).[14]
Domain-specific evidence is strongest for pain, IBS, hot flashes, and procedural medicine. Gut-directed hypnotherapy — typically taught with self-hypnosis for home practice — is recommended in European and North American clinical guidelines for IBS.[8] A well-conducted RCT of clinical hypnosis (including self-hypnosis instruction) for postmenopausal hot flashes produced statistically significant reductions in frequency and severity (N=187).[15] Evidence for depression is meaningfully weaker: a 2024 systematic review with meta-analysis concluded insufficient evidence to indicate hypnosis-based interventions reduce depression severity.[16]
| RCTs in Elkins 2018 self-hypnosis review | 22 of 576 screened |
| Meta-analyses in Rosendahl 2024 synthesis | 49 covering 261 primary studies |
| Medium-or-larger effects in Rosendahl 2024 | ~54% (25.4% medium, 28.8% large) |
| Effect size on surgical pain (Tefikow 2013) | g=0.44 |
| Effect size on emotional distress (Tefikow 2013) | g=0.53 |
| High-hypnotizable population fraction | ~10–15% |
Limitations
Several limitations qualify the evidence base.
Heterogeneity of intervention
Most meta-analyses pool clinician-administered (heterohypnosis) and self-administered hypnosis without distinguishing them, making domain-general effect-size estimates only loosely applicable to self-practice. The direct self-hypnosis evidence base — 22 RCTs in Elkins 2018 — is narrower.[1]
Methodological quality
The Rosendahl 20-year review applied AMSTAR 2 criteria to its 49 included meta-analyses and rated only 9 of them as high methodological quality.[13] True blinding is methodologically difficult in hypnosis research, and control conditions vary substantially across trials — attention placebo, structured relaxation, treatment-as-usual — making cross-study synthesis sensitive to choice of comparator.
Unaccounted moderation by hypnotizability
Trait hypnotizability is a significant moderator of hypnotic response, but most trials do not stratify by it, introducing variance that attenuates observed effects. Recent translational work targeting hypnotizability directly is beginning to address this gap,[12] but the existing evidence base under-represents the variance attributable to this trait.
Weaker domains
Depression evidence is insufficient to support efficacy at present.[16] Smoking cessation evidence is mixed and a Cochrane review found insufficient evidence to determine whether hypnotherapy outperforms other behavioral support. These domains should not be conflated with the better-evidenced ones (pain, IBS, procedural medicine, hot flashes) when summarizing the field.
references
- [1]
- [2] ^abcdJiang, H. and White, M.P. and Greicius, M.D. and Waelde, L.C. and Spiegel, D. (2017) Brain Activity and Functional Connectivity Associated with Hypnosis — Cerebral Cortex doi:10.1093/cercor/bhw220
- [3]
- [4]
- [5]
- [6]